Weight loss, insulin sensitivity, human trials, side effects, FDA status, prices, and global verification

MOTS-c has a new human trial, not proven human benefits.

MOTS-c is widely sold for weight loss, energy, longevity, and exercise claims. The foundational results come from cells and rodents, while human studies mostly measure naturally circulating MOTS-c. One exact-named Phase 2a trial is now recruiting with no results posted. This guide keeps all four records separate.

Amino acids16Mitochondrial-derived peptide
Recruiting exact trial01NCT07505745
Posted trial results00For exact administered MOTS-c
FDA-approved products00Free base or acetate
Direct answer

There is now a MOTS-c intervention study, but there is still no posted human outcome.

ClinicalTrials.gov lists a recruiting 120-participant Phase 2a study of an intervention named MOTS-c in adults with prediabetes and overweight or obesity. Its first posted date is April 1, 2026. The registry has no results and does not state whether the intervention is MOTS-c free base or acetate.

FDA's May 11, 2026 briefing says it found no MOTS-c clinical study or human exposure data. The two dated primary records do not currently reconcile, so this guide preserves the conflict instead of silently choosing one.

Evidence summary

Read the MOTS-c record in four fields.

This is a sourced status snapshot, not a safety score, treatment recommendation, or seller certification. Each field links back to the detailed record below.

01 Strongest human record

Recruiting Phase 2a

The exact-named 120-participant trial is recruiting and has no posted results.

Inspect the human evidence
02 What it establishes

No posted human outcome

Circulating-peptide studies and preclinical findings do not prove a treatment benefit.

Audit the popular claims
Best primary source to open firstNCT07505745 MOTS-c Phase 2a recordClinicalTrials.gov, National Library of Medicine
Open the primary record
Primary-source conflict watch

Two primary records do not currently reconcile

FDA's dated briefing says it searched ClinicalTrials.gov and found no human administration study. The NCT07505745 record says it was first posted forty days earlier and describes a recruiting Phase 2a MOTS-c intervention. The public records do not explain the mismatch. Possible differences in search timing, identity matching, record availability, or review workflow cannot be resolved from the documents alone.

May 11, 2026

FDA says no administered human record identified

The agency briefing states that its review included ClinicalTrials.gov and did not identify clinical studies or human exposure data for MOTS-c by any route.

Inspect the FDA document →
April 1, 2026

ClinicalTrials.gov posts a recruiting Phase 2a record

NCT07505745 describes randomized subcutaneous MOTS-c treatment in 120 estimated participants, with an actual February 2026 start and no results posted.

Inspect the registry record →
Peptide Local rule

Preserve both dated claims

Preserve both records. Treat the FDA conclusion as the agency's dated compounding review and the registry as a current study plan with no results. Do not convert either into proof that treatment works or is safe.

Read the evidence method →
Five evidence lanes

Biology, biomarkers, intervention trials, and approval answer different questions.

The strongest useful answer preserves the exact intervention and study stage. Endogenous MOTS-c in a blood sample, synthetic MOTS-c in a mouse, CB4211 in a person, and a seller vial are not interchangeable.

01Identity

MOTS-c is a 16-amino-acid mitochondrial-derived peptide

FDA evaluates MOTS-c free base and MOTS-c acetate as distinct bulk drug substances. The exact form, sequence, formulation, route, and finished product still have to match before evidence can transfer to a vial or claim.

02Preclinical evidence

Weight and glucose signals come mainly from cells and rodents

The foundational record includes AMPK-linked mechanisms and mouse findings involving diet-induced obesity and insulin resistance. FDA says dose-response information is incomplete and the clinical relevance of these nonclinical findings remains unknown.

03Human measurement

People have been studied as a source of endogenous MOTS-c data

Human exercise, metabolic, and cardiovascular studies measure MOTS-c already circulating in blood or present in tissue. Those studies can describe association or physiology, but they do not show that injecting synthetic MOTS-c causes weight loss or improves health.

04Human intervention

One new exact-named Phase 2a trial is recruiting

ClinicalTrials.gov lists NCT07505745 as a 120-participant recruiting study of subcutaneous MOTS-c for insulin sensitivity in adults with prediabetes and overweight or obesity. No results are posted, and the registry does not specify free base versus acetate.

05Regulatory record

No FDA-approved MOTS-c product

FDA staff proposed not adding MOTS-c free base or acetate to the 503A Bulks List. On July 23, PCAC recommended inclusion after reported 7-5-2 votes on the paired form questions. That advice is nonbinding and does not itself approve a drug, immediately change the list, or finally decide either substance.

Claim check

What the MOTS-c record shows, and what it cannot prove.

Each row keeps the signal, exact evidence type, and missing inference together.

Popular claimActual recordEvidence limit

MOTS-c causes weight loss in people

Foundational studies reported lower weight gain and improved metabolic measures in high-fat-fed mice. A new 120-participant Phase 2a registry record includes body weight and metabolic outcomes.

The mouse results are not human treatment results. NCT07505745 is recruiting and has no posted outcomes, so the current record cannot establish average weight loss, responder rates, durability, or comparison with approved obesity medicines.

MOTS-c improves insulin sensitivity

Cell and animal research provides a mechanistic and preclinical signal. Human studies also report associations between endogenous circulating MOTS-c and metabolic measures.

An endogenous biomarker association does not show that an administered product produces the same effect. The recruiting Phase 2a trial is designed to test insulin sensitivity, but it has not posted results.

Exercise proves MOTS-c works as a performance peptide

Human studies have measured changes in circulating or skeletal-muscle MOTS-c around endurance and resistance exercise conditions.

Exercise-induced physiology is not an administration trial. Measuring a naturally occurring peptide after exercise does not establish that an injected research product improves strength, endurance, recovery, body composition, or performance.

The CB4211 human trial proves MOTS-c safety

NCT03998514 studied CB4211 in 88 participants. The sponsor described CB4211 in an SEC filing as an improved MOTS-c analog.

An analog is not the exact native MOTS-c sequence or necessarily the same active moiety, pharmacokinetics, formulation, impurity profile, or risk. The registry also has no posted results, so it cannot supply a public event rate here.

No reported side effects means MOTS-c is safe

FDA's FAERS searches through March 9, 2025 retrieved no MOTS-c adverse-event reports, while the agency also found no administered human exposure data in its review.

No report is not a safety study. FDA highlights unresolved immunogenicity, aggregation, peptide-related impurity, endotoxin, formulation, route, sterility, and characterization concerns, and event rates remain unknown.

Human evidence ledger

One FDA review, one recruiting treatment trial, two biomarker records, and one analog trial.

Only NCT07505745 lists an exact-named MOTS-c treatment intervention. It has no posted result. Every other row has a boundary that prevents it from proving the same claim.

FDA 2026 reviewNo human exposure identified in the briefing

FDA search for administered MOTS-c evidence

Identity
MOTS-c free base or MOTS-c acetate
Design
Regulatory literature, trial-registry, safety, and chemistry review
Participants
0 identified by FDA

The May 11 FDA briefing says the agency did not identify a clinical study or human exposure record for either reviewed bulk drug substance. It therefore could not estimate pharmacokinetics, effectiveness, adverse-event frequency, or long-term safety.

NCT07505745Recruiting, no results posted

MOTS-MET insulin-sensitivity trial

Identity
Intervention named MOTS-c; free base or acetate not stated
Design
Phase 2a, randomized, double-blind, placebo-controlled
Participants
120 estimated

ClinicalTrials.gov first posted this record on April 1, 2026. It lists 12 weeks of subcutaneous treatment in adults with prediabetes and overweight or obesity, followed through week 16 for safety. A recruiting registry record is a study plan, not evidence of benefit or safety.

NCT04027712Observational, no MOTS-c intervention

MOTS-c as a cardiovascular biomarker

Identity
Endogenous circulating MOTS-c measurement
Design
Prospective observational follow-up
Participants
120 estimated

This study measures naturally circulating MOTS-c alongside platelet reactivity and cardiovascular outcomes in people with type 2 diabetes and coronary artery disease. It can test association, but it cannot establish the effects of administering synthetic MOTS-c.

PMID 34351816Human physiology, no MOTS-c treatment

Acute exercise and endogenous MOTS-c

Identity
Plasma and skeletal-muscle MOTS-c measurement
Design
Randomized endurance, resistance, or control experiment
Participants
30

The investigators measured MOTS-c before and after controlled exercise conditions. This helps explain human physiology, but does not test a seller vial, an injected dose, weight loss, recovery, or clinical treatment benefit.

NCT03998514Completed, no registry results posted

CB4211 in healthy participants and NAFLD

Identity
CB4211, described by its sponsor as a MOTS-c analog
Design
Completed Phase 1a/1b randomized, placebo-controlled study
Participants
88 actual

This is a real human administration record for a related development candidate, not direct evidence for exact MOTS-c free base or acetate. Molecule identity and public results must match before safety or efficacy findings can transfer.

Evidence-market gap

The research market is already larger than the completed human-result record.

Public availability is a market fact, not clinical validation. These figures describe dated 10 mg US seller pages and exclude coupons, checkout-only discounts, shipping, and unconfirmed rows from the in-stock range.

Qualified exact-size rows

07

Seven public US seller pages met the current 10 mg comparison rules. Inclusion does not mean approval, recommendation, product authentication, or provider certification.

Open every source row →
Available rows

05

Four rows carried an in-stock signal when checked. Availability can change and does not establish legality, fulfillment, composition, sterility, or destination access.

Check source freshness →
In-stock list-price range

$37.00-$99.00

The current in-stock range spans 168% from low to high before landed cost. Compare documentation and availability beside price, then verify the exact product and seller.

Compare landed cost →
Current FDA process

The July 23 recommendation is meaningful, but it is not approval or final action.

FDA staff analysis, advisory committee discussion, and final agency action are separate stages.

Current FDA staff proposal

Do not add either MOTS-c form

FDA proposes not adding MOTS-c free base or MOTS-c acetate to the 503A Bulks List after weighing characterization, historical use, effectiveness, safety, and available alternatives.

Read the briefing →
July 23, 2026

Favorable committee recommendation

PCAC recommended inclusion of both reviewed forms after reported 7-5-2 votes. The advice is nonbinding, does not approve MOTS-c, and does not immediately change the 503A Bulks List.

Open the verified vote record →
Later step

Final FDA determination

FDA can complete its review after considering advisory input. Until a later agency record is published, the current position remains a proposal.

Track the full process →
Worldwide answer

MOTS-c product status and access depend on the destination.

Health Canada currently names MOTS-C among seized unauthorized injectable peptide drugs. That is an important Canadian product finding, but it does not decide approval, prescription, import, compounding, seller, or product status in every other jurisdiction.

Direct evidence answers

Eleven MOTS-c questions, answered from the current record.

This is educational reporting, not medical advice, a prescription, or a use protocol.

01

What is MOTS-c?

MOTS-c, short for mitochondrial open reading frame of the 12S rRNA-c, is a 16-amino-acid mitochondrial-derived peptide. FDA separately evaluates MOTS-c free base and MOTS-c acetate as distinct bulk drug substances. A product label using the common name does not establish which form is present, exact sequence, identity, purity, aggregation, endotoxin level, sterility, formulation, storage integrity, or vial contents.

02

What does MOTS-c do?

The research record links endogenous MOTS-c to cellular energy and metabolic signaling, with AMPK-related pathways commonly discussed. Synthetic MOTS-c produced metabolic, weight, bone, and cardiovascular signals in cell and rodent models reviewed by FDA. Those mechanisms and animal findings generate hypotheses. They do not establish what an administered product does in people, how large an effect would be, or whether benefits exceed risks.

03

Does MOTS-c help with weight loss?

No completed controlled human result cited here establishes MOTS-c weight loss. Mouse studies reported less weight gain under specific experimental conditions, but FDA says the clinical relevance is unknown. NCT07505745 is now recruiting adults with prediabetes and overweight or obesity and includes body-weight outcomes, but it has no posted results. A planned endpoint is not a measured effect and cannot support a pounds-lost or percentage-lost claim.

04

Does MOTS-c improve insulin sensitivity?

Preclinical work and human biomarker studies provide a plausible metabolic signal, but they answer different questions. Measuring naturally circulating MOTS-c and finding an association with insulin-related measures does not prove that administering synthetic MOTS-c improves insulin sensitivity. The recruiting MOTS-MET Phase 2a trial is designed to test an oral-glucose-tolerance-derived insulin-sensitivity endpoint, and no outcome has been posted.

05

Has MOTS-c been tested in humans?

The most accurate current answer needs two dates. FDA's May 11, 2026 briefing says it found no MOTS-c clinical study or human exposure. ClinicalTrials.gov says NCT07505745 was first posted April 1, 2026 and is recruiting 120 participants for a Phase 2a study of an intervention named MOTS-c. That trial has no posted results, and its registry does not specify free base versus acetate. Human biomarker studies are not administration studies.

06

What are the side effects of MOTS-c?

A dependable MOTS-c side-effect profile or event rate cannot yet be calculated from completed exact-intervention results. FDA found no administered human exposure in its review and says potential human risks are unknown. The agency highlights immunogenicity, aggregation, peptide-related impurities, active-ingredient characterization, formulation, route, endotoxin, sterility, and product-quality concerns. A recruiting trial's safety endpoint does not supply results before data are posted.

07

Is MOTS-c FDA approved?

No FDA-approved MOTS-c drug is established by the cited records. FDA states that neither MOTS-c free base nor MOTS-c acetate is a component of an FDA-approved drug. FDA staff proposed non-inclusion, but PCAC recommended inclusion after reported 7-5-2 votes on July 23, 2026. That nonbinding advice does not immediately change the 503A Bulks List, approve a drug, or decide another jurisdiction.

08

Is there a MOTS-c Phase 2 trial?

Yes, ClinicalTrials.gov lists NCT07505745 as a recruiting Phase 2a randomized, double-blind, placebo-controlled trial with an estimated 120 adults. The record describes daily subcutaneous treatment for 12 weeks and follow-up through week 16, with insulin sensitivity and treatment-emergent adverse events among primary outcomes. No results are posted. Registration, recruitment, estimated enrollment, and planned endpoints do not show completion or success.

09

Is CB4211 the same as MOTS-c?

No. The CB4211 sponsor described it in a U.S. Securities and Exchange Commission filing as an improved analog of naturally occurring MOTS-c. NCT03998514 studied CB4211 in 88 participants, but an analog can differ in sequence, active moiety, exposure, pharmacokinetics, formulation, and risk. That trial therefore cannot be presented as direct human evidence for MOTS-c free base, MOTS-c acetate, or a seller-labelled MOTS-c vial.

10

What is the correct MOTS-c dosage?

There is no FDA-approved MOTS-c label or established self-use dosage for the research products discussed here. The new trial registry describes a fixed once-daily regimen but does not publicly state the dose in the summarized intervention record. Animal amounts, seller instructions, forum schedules, CB4211 doses, or unverified clinic protocols should not be converted into a personal regimen. This guide does not provide reconstitution, injection, cycling, dosing, or stacking instructions.

11

Can you buy MOTS-c legally?

The answer depends on the exact product, intended use, destination, import route, medicine authorization, pharmacy rules, professional status, and seller record. Health Canada specifically names MOTS-C among seized unauthorized injectable peptide drugs and warns that research-use-only wording does not create an exemption. That Canadian finding does not decide another country's law. Use the linked country and local regulator routes rather than relying on a seller's shipping claim.

Critical boundary

Endogenous biology is not a finished product.

A naturally circulating peptide, synthetic free base, acetate salt, analog, animal intervention, registered trial product, compounded preparation, and online research vial can differ in identity, exposure, impurities, formulation, and risk. The exact intervention must match before a result transfers.

FDA approval, 503A list status, country access, seller availability, documentation, and product authentication are separate records. None supplies an individual treatment recommendation.

Primary referencesFDA review of MOTS-c free base and MOTS-c acetateMOTS-c identity, reviewed obesity and osteoporosis uses, nonclinical evidence limits, missing human exposure record, safety concerns, and the current 503A Bulks List proposalU.S. Food and Drug AdministrationJuly 23-24, 2026 Pharmacy Compounding Advisory Committee meetingOfficial day-one broadcast route, MOTS-c agenda placement, public briefing documents, and the boundary between an FDA proposal, committee advice, and later final agency actionU.S. Food and Drug AdministrationDay-one peptide compounding vote resultsContemporaneous reporting of the favorable 7-5-2 committee recommendation for the paired MOTS-c form questions and the nonbinding status of that adviceFierce PharmaFDA bulk-substance safety risk summaryCurrent FDA summary of missing human exposure data and unresolved immunogenicity, peptide-related impurity, aggregation, active-ingredient characterization, and route concernsU.S. Food and Drug AdministrationNCT07505745 MOTS-MET Phase 2a registry recordRecruiting status, 120-participant estimate, subcutaneous intervention named MOTS-c, prediabetes and overweight or obesity population, February 2026 start, and absence of posted resultsClinicalTrials.gov, National Library of MedicineNCT04027712 observational MOTS-c biomarker studyProspective observational design, estimated enrollment of 120, endogenous circulating MOTS-c measurement, cardiovascular follow-up, and the absence of a MOTS-c treatment interventionClinicalTrials.gov, National Library of MedicineLipids and insulin regulate circulating MOTS-c in PCOS and healthy subjectsHuman physiology study measuring endogenous plasma MOTS-c during lipid and insulin exposure and after exercise, without administering synthetic MOTS-c as a treatmentPubMed, National Library of MedicineAcute endurance exercise and circulating mitochondrial-derived peptidesThirty-participant exercise experiment measuring plasma and skeletal-muscle MOTS-c around endurance, resistance, and control conditions, not therapeutic MOTS-c administrationPubMed, National Library of MedicineMOTS-c discovery and metabolic homeostasis studyThe 16-amino-acid mitochondrial peptide identity, proposed AMPK-linked mechanism, cell experiments, and obesity and insulin-resistance findings from mouse models rather than treated peoplePubMed, National Library of MedicineNCT03998514 CB4211 Phase 1a/1b registry recordCompleted 88-participant study design, subcutaneous CB4211 intervention, NAFLD population, safety and pharmacokinetic outcomes, and the absence of posted registry resultsClinicalTrials.gov, National Library of MedicineCohBar SEC filing describing CB4211 as a MOTS-c analogSponsor description of CB4211 as an improved analog of MOTS-c, establishing why CB4211 human exposure cannot be relabeled as direct evidence for the exact MOTS-c interventionU.S. Securities and Exchange CommissionHealth Canada warning on injectable peptides bought onlineApril 2026 Canadian warning naming MOTS-C among seized unauthorized injectable peptide drugs and explaining product, ingredient, contamination, storage, and licensed-pharmacy risksHealth Canada